Ó³»­´«Ã½

Thakore PI, Schnell A, Huang L, et al. BACH2 regulates diversification of regulatory and proinflammatory chromatin states in T17 cells. Nature immunology. 2024. doi:10.1038/s41590-024-01901-1

FinnGen provides genetic insights from a well-phenotyped isolated population. Nature. 2023. doi:10.1038/s41586-022-05473-8

Mono- and biallelic variant effects on disease at biobank scale. Nature. 2023. doi:10.1038/s41586-022-05420-7

Nauffal V, Morrill VN, Jurgens SJ, et al. Monogenic and Polygenic Contributions to QTc Prolongation in the Population. Circulation. 2022;145(20):1524-1533. doi:10.1161/CIRCULATIONAHA.121.057261

Damotte V, van der Lee SJ, Chouraki V, et al. Plasma amyloid β levels are driven by genetic variants near APOE, BACE1, APP, PSEN2: A genome-wide association study in over 12,000 non-demented participants. Alzheimer’s & dementia : the journal of the Alzheimer’s Association. 2021;17(10):1663-1674. doi:10.1002/alz.12333

Sherva R, Gross A, Mukherjee S, et al. Genome-wide association study of rate of cognitive decline in Alzheimer’s disease patients identifies novel genes and pathways. Alzheimer’s & dementia : the journal of the Alzheimer’s Association. 2020;16(8):1134-1145. doi:10.1002/alz.12106

Tanskanen T, van den Berg L, Välimäki N, et al. Genome-wide association study and meta-analysis in Northern European populations replicate multiple colorectal cancer risk loci. Int J Cancer. 2018;142(3):540-546. doi:10.1002/ijc.31076

Yu L, Lutz MW, Farfel JM, et al. Neuropathologic features of TOMM40 ’523 variant on late-life cognitive decline. Alzheimers Dement. 2017;13(12):1380-1388. doi:10.1016/j.jalz.2017.05.002

Felsky D, Xu J, Chibnik LB, et al. Genetic epistasis regulates amyloid deposition in resilient aging. Alzheimers Dement. 2017;13(10):1107-1116. doi:10.1016/j.jalz.2017.01.027

Jun GR, Chung J, Mez J, et al. Transethnic genome-wide scan identifies novel Alzheimer’s disease loci. Alzheimers Dement. 2017. doi:10.1016/j.jalz.2016.12.012