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Publications

Template-assisted covalent modification of DCAF16 underlies activity of BRD4 molecular glue degraders.

Der Li Y, Ma MW, Hassan MM, et al. Template-assisted covalent modification of DCAF16 underlies activity of BRD4 molecular glue degraders. bioRxiv : the preprint server for biology. 2023. doi:10.1101/2023.02.14.528208

Shared and distinct ultra-rare genetic risk for diverse epilepsies: A whole-exome sequencing study of 54,423 individuals across multiple genetic ancestries.

Chen S, Neale BM, Berkovic SF, . Shared and distinct ultra-rare genetic risk for diverse epilepsies: A whole-exome sequencing study of 54,423 individuals across multiple genetic ancestries. medRxiv : the preprint server for health sciences. 2023. doi:10.1101/2023.02.22.23286310

Developing a Phenotype Risk Score for Tic Disorders in a Large, Clinical Biobank.

Miller-Fleming TW, Allos A, Gantz E, et al. Developing a Phenotype Risk Score for Tic Disorders in a Large, Clinical Biobank. medRxiv : the preprint server for health sciences. 2023. doi:10.1101/2023.02.21.23286253

Distinct and shared genetic architectures of Gestational diabetes mellitus and Type 2 Diabetes Mellitus.

Elliott A, Walters RK, Pirinen M, et al. Distinct and shared genetic architectures of Gestational diabetes mellitus and Type 2 Diabetes Mellitus. medRxiv : the preprint server for health sciences. 2023. doi:10.1101/2023.02.16.23286014

Template-assisted covalent modification of DCAF16 underlies activity of BRD4 molecular glue degraders.

Der Li Y, Ma MW, Hassan MM, et al. Template-assisted covalent modification of DCAF16 underlies activity of BRD4 molecular glue degraders. bioRxiv : the preprint server for biology. 2023. doi:10.1101/2023.02.14.528208

Shared and distinct ultra-rare genetic risk for diverse epilepsies: A whole-exome sequencing study of 54,423 individuals across multiple genetic ancestries.

Chen S, Neale BM, Berkovic SF, . Shared and distinct ultra-rare genetic risk for diverse epilepsies: A whole-exome sequencing study of 54,423 individuals across multiple genetic ancestries. medRxiv : the preprint server for health sciences. 2023. doi:10.1101/2023.02.22.23286310

Developing a Phenotype Risk Score for Tic Disorders in a Large, Clinical Biobank.

Miller-Fleming TW, Allos A, Gantz E, et al. Developing a Phenotype Risk Score for Tic Disorders in a Large, Clinical Biobank. medRxiv : the preprint server for health sciences. 2023. doi:10.1101/2023.02.21.23286253

Distinct and shared genetic architectures of Gestational diabetes mellitus and Type 2 Diabetes Mellitus.

Elliott A, Walters RK, Pirinen M, et al. Distinct and shared genetic architectures of Gestational diabetes mellitus and Type 2 Diabetes Mellitus. medRxiv : the preprint server for health sciences. 2023. doi:10.1101/2023.02.16.23286014

Shared and distinct ultra-rare genetic risk for diverse epilepsies: A whole-exome sequencing study of 54,423 individuals across multiple genetic ancestries.

Chen S, Neale BM, Berkovic SF, . Shared and distinct ultra-rare genetic risk for diverse epilepsies: A whole-exome sequencing study of 54,423 individuals across multiple genetic ancestries. medRxiv : the preprint server for health sciences. 2023. doi:10.1101/2023.02.22.23286310

Developing a Phenotype Risk Score for Tic Disorders in a Large, Clinical Biobank.

Miller-Fleming TW, Allos A, Gantz E, et al. Developing a Phenotype Risk Score for Tic Disorders in a Large, Clinical Biobank. medRxiv : the preprint server for health sciences. 2023. doi:10.1101/2023.02.21.23286253